Dr. Mahmoud Naas

Research Grant Manager

Email

mnaas@hbku.wdu.qa

Phone

66 980 238

Office location

Office # 1360 Segment 6, Building 2 HBKU Research Complex

Dr. Mahmoud Naas

Research Grant Manager

Educational Qualifications

PhD

MSc

Entity

Qatar Biomedical Research Institute

Division

Management

Biography

Mahmoud Naas has been in the medical research field and academia for more than 30 years. .

He was previously  an associate professor at Qatar University’sCollege of Health Sciences, where he  developed and delivered a variety of courses. 

As a scientist, Professor Naas’ research activity is an integral part of his academic role; his research has generated several peer reviewed publications, as well as poster presentations at national and international conferences.

 

PhD

Reading - United Kingdom

1992

MSc

Reading - United Kingdom

1986

BSc

The Higher Institute of Technology, Brack - Libya

1983

  • The development of new method for Haemoglobinopathy Screening –all single
  • Non-invasive early pregnancy screening for maternal and fetal wellbeing
  • such as Preeclampsia
  • Ectopic Pregnancy
  • Gestational Diabetes and Fetal Aneuploidy T21 Down Syndrome.
  • Cancer-Early diagnosis and monitoring.

Associate Professor

Community College Qatar (CCQ), Doha - Qatar

2018 - 2019

Project Manager

Map Sciences Ltd, Bedford - United Kingdom

2016 - 2018

Associate Professor

Qatar University, Doha - Qatar

2013 - 2016

Senior Lecturer

Middlesex University, London _ United Kingdom

2004 - 2013

Tissue Bank Manager

Queen Mary University, London – UK

2001 - 2004

Future Laboratory Medicine: Rapid, Efficient and Affordable Screening for Haemoglobinopathies by MALDI-ToF Mass Spectrometry. Adv Biochem Biotehcnol (in press)

Novel insights into the expression of CGB1 & 2 genes by epithelial cancer cell lines secreting ectopic free hCGβ. Anticancer Research 34:2239-2248

No evidence for the involvement of XMRV or MCV in the pathogenesis of breast cancer British Journal of Cancer 106:1166 – 1170

Human Papilloma Viruses Infections in Sporadic Benign Prostatic Hyperplasia and Prostate Cancer Journal of Pathology 226: S14-

GLI1 induces a basal-like phenotype that confers androgen independence upon LNCaP prostate cancer cells. PLoS One. ;6 (5)

Association between Large-scale Genomic Homozygosity without Chromosomal Loss and Nonseminomatous Germ Cell Tumor Development. Cancer Res 65: 9137-9141

  • 1997; Research Fellowship; Joint Research Board, St Bartholomew’s Hospital; London – United Kingdom