قدِّم طلبك الآن
الدكتور محمود ناس
مدير المنح البحثية
البريد الإلكتروني
mnaas@hbku.wdu.qaالهاتف
66 980 238موقع المكتب
Office # 1360 Segment 6, Building 2 HBKU Research Complex
الدكتور محمود ناس
مدير المنح البحثية
المؤهلات العلمية
PhD
MSc
الكيان
معهد قطر لبحوث الطب الحيوي
القسم
الإدارة
السيرة الذاتية
Mahmoud Naas has been in the medical research field and academia for more than 30 years. .
He was previously an associate professor at Qatar University’sCollege of Health Sciences, where he developed and delivered a variety of courses.
As a scientist, Professor Naas’ research activity is an integral part of his academic role; his research has generated several peer reviewed publications, as well as poster presentations at national and international conferences.
PhD
Reading - United Kingdom
1992
MSc
Reading - United Kingdom
1986
BSc
The Higher Institute of Technology, Brack - Libya
1983
- The development of new method for Haemoglobinopathy Screening –all single
- Non-invasive early pregnancy screening for maternal and fetal wellbeing
- such as Preeclampsia
- Ectopic Pregnancy
- Gestational Diabetes and Fetal Aneuploidy T21 Down Syndrome.
- Cancer-Early diagnosis and monitoring.
Associate Professor
Community College Qatar (CCQ), Doha - Qatar
2018 - 2019
Project Manager
Map Sciences Ltd, Bedford - United Kingdom
2016 - 2018
Associate Professor
Qatar University, Doha - Qatar
2013 - 2016
Senior Lecturer
Middlesex University, London _ United Kingdom
2004 - 2013
Tissue Bank Manager
Queen Mary University, London – UK
2001 - 2004
Future Laboratory Medicine: Rapid, Efficient and Affordable Screening for Haemoglobinopathies by MALDI-ToF Mass Spectrometry. Adv Biochem Biotehcnol (in press)
Novel insights into the expression of CGB1 & 2 genes by epithelial cancer cell lines secreting ectopic free hCGβ. Anticancer Research 34:2239-2248
No evidence for the involvement of XMRV or MCV in the pathogenesis of breast cancer British Journal of Cancer 106:1166 – 1170
Human Papilloma Viruses Infections in Sporadic Benign Prostatic Hyperplasia and Prostate Cancer Journal of Pathology 226: S14-
GLI1 induces a basal-like phenotype that confers androgen independence upon LNCaP prostate cancer cells. PLoS One. ;6 (5)
Association between Large-scale Genomic Homozygosity without Chromosomal Loss and Nonseminomatous Germ Cell Tumor Development. Cancer Res 65: 9137-9141
- 1997; Research Fellowship; Joint Research Board, St Bartholomew’s Hospital; London – United Kingdom